Clen is a steroid, recommended for the victims of lung diseases, and work as a bronchodilator or chest decongestant. People suffering from asthma, us it when they went short of oxygen as its action is very immediate. It is most commonly available in salt form as Clenbuterol hydrochloride.

Clen is a ?2 adrenergic agonist in formulation, which closely resemble to another drug ephedrine, but it is more thermogenic and long acting stimulant. It accelerates the metabolic rate of the body that force more oxygen intake and degenerate the stored fats of the body.

This steroid stimulate the central nervous system (CNS), in the result of this stimulation CNS increase the rate of heart beat so blood starts to circulate more faster, causes an increase in aerobic capacity.

It increases the consumption of body fat and protein and simultaneously slows down the glycogen storage. It has very excellent smooth muscle relaxant this is the reason of its wide use in lung ailments. You should get complete detail of its all pros and cons before you buy Clenbuterol.

Fortuitously some people, soon after it development, discovered that taking this drug reduce the body fats and increase mass and strength of the muscles. Clenbuterol is highly anabolic drug so its both effects can be seen right after the use.

Effects that don’t last long of clen includes after its use you would feel overheated, nauseous, anxious and some time dehydrated. The longer term side effects are heart failure, enlargement of left ventricle. This drug, if abused can severely damage your eyes as well.

Recommended dose of clen is 20 to 60 micrograms each day and should not exceed from 200 micrograms.

A critical analysis of nine randomized trials, including almost 70,000 hypertensive patients, illustrates that reduction in heart rate due to beta-blockers, increases the risks of all-cause death, cardiovascular death, MI, stroke, and heart failure (p < 0.0001 for all).

Dr. Franz H. Messerli, from Columbia University, said that “pharmacologically induced bradycardia may lead to dyssynchrony between outgoing and reflected pulse wave, thereby increasing central aortic pressure and the hemodynamic burden to the target organs.”

A total of 22 randomized controlled trials were recognized, of which 9 included heart rate data. All told, 34,096 patients received beta-blockers, 30,139 received other antihypertensive drugs, and 3987 received placebo.

Patients treated beta-blocker, attained lower heart rates than did other patients, this heart rate lowering was linked to adverse cardiovascular events. Moreover, the slower the heart rate, the greater the risk of cardiovascular diseases.

In an accompanying editorial, Dr. Norman M. Kaplan, from the University of Texas, comments that the present research “adds another post-mortem explanation for the fall of beta-blockers, showing higher mortality associated with the slower heart rate they induce.”

He adds that “with this addition to the evidence, beta-blockers will surely remain as indicated for heart failure, for after MI, and for tachyarrhythmias, but no longer for hypertension in the absence of these compelling indications.”

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