NEW YORK – Being obese may faint a man’s hope of becoming a father, even if he is healthy superficially, a new study concludes.

Researchers came to know that among 87 healthy men ages 19 to 48, obese were less likely to be a father. Precisely, they exhibit hormonal variation that leads to a reduced reproductive capacity, the researchers report in the journal Fertility and Sterility.

As compare to thinner men, obese had reduced levels of testosterone in their blood, as well as poorer levels of luteinizing hormone (LH) and follicle-stimulating hormone (FSH), both hormones are essential for reproduction.

Researchers said, these comparatively low levels of LH and FSH are indicative of a “partial” hypogonadotropic hypogonadism, a condition in which the testes do not work appropriately due to weak signals from hypothalamus or pituitary gland, two important parts of brain involved in hormone secretion for endocrine system.

Researchers suggest that obesity alone is a key “infertility factor” in so-called healthy men, write Dr. Eric M. Pauli.

When the researchers examined the men for various reproductive hormones, they confirm that the LH and FSH levels are inversely proportional to the obesity. On the other side, increasing obesity can be associated with elevating estrogen levels.

Pauli’s team explained that, excessive body fats increase the conversion of testosterone to estrogen in a man’s blood. Such hormone alterations could signal the brain to reduce or even stop FSH and LH production.

Previous studies have linked obesity with a dampened libido and higher risk of erectile dysfunction, the researchers note. Those effects along with the hormonal alterations seen in this study, could act together to decrease an obese man’s fertility, researcher said.

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